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We investigate the age and structure of the human clade, with special reference to the fossil record. This includes formal hypotheses of how and why our clade has been demographically successful and what characters are unique to it.

We investigate how metabolism (especially autophagy and nitrogen homeostasis) interfaces with sleep and circadian rhythms, using behavioral, imaging, and biochemical approaches.

We investigate why some people are better than others at perceiving, and learning to perceive, basic auditory and speech sounds.

We investigate the use of nanotechnology for the treatment of diseases including multidrug-resistant bacterial infections, non-healing chronic wounds, and Alzheimer’s disease.

We investigate cellular and molecular mechanisms responsible for accelerated vascular complications in diabetes and obesity. We also study the role of cerebrovascular dysfunction in Alzheimer’s disease-related pathology and cognitive impairment in metabolic disorders.

My lab focuses on the locomotor function of the mammalian head and neck and how their functions may influence craniovertebral form.

We develop and apply different 3D imaging and computational techniques to understand diseases of the brain, for virtual and physical reconstruction as well as education.

We investigate the potential interactions that exist between biomechanics and neurophysiology. Her ultimate goals are to (1) enhance our knowledge of dysfunction as the basis for developing rehabilitation strategies, particularly for children and (2) explicate the evolutionary basis of mammalian feeding functional morphology.

We investigate the disease mechanisms underlying myocardial infarction and heart failure using rodent models. Our research focuses on how mitochondrial dysfunction, caused by oxygen toxicity and free radical injury, contributes to the pathophysiology of heart disease.

We investigate the therapeutic potential of neuromodulation-based approaches to the treatment of neurological disorders.